|Salary:||£32,236 to £39,609|
|Placed On:||18th January 2019|
|Closes:||18th February 2019|
Circadian clocks govern nearly all aspects of mammalian physiology and behaviour. We have characterized cell autonomous circadian clocks in articular cartilage, which control key pathways in tissue homeostasis. Importantly, we have shown that the cartilage circadian rhythm and expression of chondrocyte clock genes change profoundly with ageing and during early stages of osteoarthritis initiation. This project aims to address how individual circadian clock genes regulate cartilage homeostasis and chondrocyte biology, and test the hypothesis that targeting clock genes may slow down disease progression, and promote tissue regeneration and repair. The Research Associate post is funded by Versus Arthritis. You are expected to be a highly motivated scientist who is excited to be part of a translational research project that involves patients, clinical samples and pharmacological compounds. Evidence of conducting research to a high standard, and publishing results in leading peer-reviewed journals, are essential.
The School is strongly committed to promoting equality and diversity, including the Athena SWAN charter for gender equality in higher education. The School holds a Silver Award which recognises their good practice in relation to gender; including flexible working arrangements, family-friendly policies, and support to allow staff achieve a good work-life balance. We particularly welcome applications from women for this post. Appointment will always be made on merit. For further information, please visit: https://www.bmh.manchester.ac.uk/about/equality/
Please note that we are unable to respond to enquiries, accept CVs or applications from Recruitment Agencies
Enquiries about vacancy shortlisting and interviews:
Name: Qing-Jun Meng.
Tel: 0161 306 8912
Tel: 0161 275 4499
Tel: 0161 850 2004
This vacancy will close for applications at midnight on the closing date
Type / Role: